The Netter Collection of Medical Illustrations, Volume 6, Part 3: Musculoskeletal System (2nd Edition)
Joseph P. Iannotti, Richard D. Parker
Language: English
Pages: 357
ISBN: 2:00277631
Format: PDF / Kindle (mobi) / ePub
Basic Science and Systemic Disease, Part 3 of The Netter Collection of Medical Illustrations:Musculoskeletal System, 2nd Edition, provides a highly visual guide to this body system, from foundational basic science and anatomy to orthopaedics and rheumatology. This spectacularly illustrated volume in the masterwork known as the (CIBA) "Green Books" has been expanded and revised by Dr. Joseph Iannotti, Dr. Richard Parker, and other experts from the Cleveland Clinic to mirror the many exciting advances in musculoskeletal medicine and imaging - offering rich insightsinto embryology; physiology; metabolic disorders; congenital and development disorders; rheumatic diseases; tumors of musculoskeletal system; injury to musculoskeletal system; soft tissue infections; and fracture complications.
• Get complete, integrated visual guidance on the musculoskeletal system with thorough, richly illustrated coverage.
• Quickly understand complex topics thanks to a concise text-atlas format that provides a context bridge between primary and specialized medicine.
• Clearly visualize how core concepts of anatomy, physiology, and other basic sciences correlate across disciplines.
• Benefit from matchless Netter illustrations that offer precision, clarity, detail and realism as they provide a visual approach to the clinical presentation and care of the patient.
• Gain a rich clinical view of embryology; physiology; metabolic disorders; congenital and development disorders; rheumatic diseases; tumors of musculoskeletal system; injury to musculoskeletal system; soft tissue infections; and fracture complications in one comprehensive volume, conveyed through beautiful illustrations as well as up-to-date radiologic and laparoscopic images.
• Benefit from the expertise of Drs. Joseph Iannotti, Richard Parker, and esteemed colleagues from the Cleveland Clinic, who clarify and expand on the illustrated concepts.
• Clearly see the connection between basic science and clinical practice with an integrated overview of normal structure and function as it relates to pathologic conditions.
• See current clinical concepts in orthopaedics and rheumatology captured in classic Netter illustrations, as well as new illustrations created specifically for this volume by artist-physician Carlos Machado, MD, and others working in the Netter style.
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Indications for therapy in postmenopausal women and men over age 50 1. After hip or spine fracture 2. BMD T-score in spine or proximal femur Յ–2.5 3. BMD between –1 and –2.5 and one of the following: a. 10-year risk of major fracture of 20% or more* b. 10-year risk of hip fracture of 3% or more* * Calculated by FRAX® algorithm Based on the 2008 Guidelines from the National Osteoporosis Foundation â•… most effective prior to attainment of peak bone mass especially combined with calcium intake.
sequence COL1A1 and COL1A2 genes as an initial diagnostic tool. Analysis of radiolabeled fibroblast collagens still has diagnostic value in selected clinical contexts. Management of OI focuses on therapies to minimize fractures and deformities, minimize disability, and promote overall health. Orthopedic management is the mainstay of treatment, along with physical therapy and exercise. In infants, the use of soft bandages is generally adequate for the management of fractures; in children and
muscle, it can be assumed that the muscles are subdivisions of an original myotome. Thus, the developmental histories of adult muscles formed by early fusion, splitting, migration, or other modifications can be reconstructed with considerable certainty. Example: knee joint Example: sternoclavicular joint Nearly all the skeletal muscles are present and, in essence, have their mature form in a fetus of 8 weeks with a crown-to-rump length of about 30╯mm (see Plate 1-16). From the time the first
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kidney, and bone. FGF-23, synthesized by the osteocyte, is a major regulator of renal phosphate reabsorption as well as PTH synthesis. Preliminary data suggest that FGF-23 also has effects on osteoid mineralization. FGF-23 increases early in CKD, much earlier than PTH, and is the peptide responsible for the phosphaturia and inhibition of 1,25(OH)2D levels in the syndrome of oncogenic osteomalacia. The information presented in Plates 3-5 and 3-12 puts into perspective the links between